Anyone who recalls his childhood wounds has probably heard many times ‘Don't pick that scab, you'll just make it worse!’.
Tissue damage initiates a rapid blood clotting, and injuries are healed by the creation of new epidermal cells.
When you eliminate a scab, you are also eliminating some of the newly regenerated tissues growing underneath, thereby interfering with the healing mechanism.
What is Wound Healing?
A tissue injury via an incision is generally followed by bleeding.
The process of vasoconstriction and coagulation starts with clotted blood immediately impregnating the wound, leading to hemostasis, and after dehydration, a scab appears.
The arrival of inflammatory cells follows, with the liberation of cellular substances and mediators.
Angiogenesis (production of blood vessels) and re-epithelization occurs and the deposition of fresh cellular and extracellular elements ensues.
The Injury Healing Mechanism in detail
Injury healing is an intricate and dynamic mechanism of reconstruction of skin cellular structures and tissue layers.
The process of wound healing is composed of various steps that lead to the formation of scar tissue in replacement of tissue that has been damaged.
The human adult wound healing process can be divided into 3 great distinct phases:
1- the inflammatory phase
2- the fibroblastic phase
3- the scar maturation phase
The inflammatory phase occurs immediately following the injury and lasts approximately 6 days.
The fibroblastic phase occurs at the termination of the inflammatory phase and can last up to 4 weeks.
Scar maturation starts at the fourth week and can last for years.
These 3 phases include various processes: chemotaxis (attraction of cells by chemical discharge), phagocytosis, neocollagenesis, collagen degradation, and collagen remodeling.
Also, angiogenesis (new cappilaries), epithelization, and the production of new glycosaminoglycans (GAGs) and proteoglycans are vital to the wound healing milieu.
The end of these biological processes results in the replacement of healthy skin structures with fibroblastic mediated scar tissue.
This process can make an exuberance of fibrous protein synthesis with a resultant hypertrophic scar, which by definition is confined to the wound area. Further exuberance can result in keloid formation where scar production extends beyond the area of the original insult. Conversely, insufficient healing can result in atrophic scar formation like in stretch marks.
How to improve the Wound Healing Mechanism?
Copper & Zinc as trace elements have a crucial role in the healing of acne lesions and in wound healing. These molecules and elements act as biological activators of both the elimination of devitalized and damaged skin cells and the rejuvenation of healthy cells. And also destroy opportunistic micro-organisms that thrive in wounds, while at the same time reconstructing the network of blood vessels that increase oxygenation and nutrition into the newly formed healthy cells.
A safer and biological option to chemical skin care solutions is now available in the shape of a skin care product to treat a wide range of skin conditions.